Annals ofAutoimmunity

Volume: 6 - Number: 57 - April 15, 2007
Archive of Annals of Autoimmunity

In this issue

Diabetes e Incretinas

Articles

Proinsulin: a unique autoantigen triggering autoimmune diabetes.

Genome-wide identification of functionally distinct subsets of cellular mRNAs associated with two nucleocytoplasmic-shuttling mammalian splicing factors.

Novel self-epitopes derived from aggrecan, fibrillin, and matrix metalloproteinase-3 drive distinct autoreactive T-cell responses in juvenile idiopathic arthritis and in health.

Role of TAB1 in nitric oxide-induced p38 activation in insulin-producing cells.

Robustness and Fragility in Immunosenescence

Direct and indirect effects of retinoic acid on human Th2 cytokine and chemokine expression by human T lymphocytes

A self-inactivating retrovector incorporating the IL-2 promoter for activation-induced transgene expression in genetically engineered T-cells.

Antigenic Differences Between Normal and Malignant Cells as a Basis for Treatment of Intracerebral Neoplasms Using a DNA-Based Vaccine

MLN51 and GM-CSF involvement in the proliferation of fibroblast-like synoviocytes in the pathogenesis of rheumatoid arthritis.

Predicting Class II MHC-Peptide binding: a kernel based approach using similarity scores

Endogenous Cell Repair of Chronic Demyelination

Original Research Article: Longitudinal Relationships of Depressive Symptoms to Pain Intensity and Functional Disability Among Children with Disease-Related Pain.

Behavioral Heterogeneity in an Animal Model of Neuropsychiatric Lupus

Elevated immunoglobulin levels in the cerebrospinal fluid from lupus-prone mice

Alterations in CD46-mediated Tr1 regulatory T cells in patients with multiple sclerosis

Differential effects on BAFF and APRIL levels in rituximab-treated patients with systemic lupus erythematosus and rheumatoid arthritis

An adult patient with new-onset dysphagia.

SuperHapten: a comprehensive database for small immunogenic compounds.

Evolution and comparative analysis of the MHC Class III inflammatory region

Interactions between IL-32 and tumor necrosis factor alpha contribute to the exacerbation of immune-inflammatory diseases

Diabetes e Incretinas

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