Annals ofVascular Medicine

Volume: 10 - Number: 106 - February 14, 2010

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Editorial

Adenine nucleotide translocator, a mitochondrial carrier protein, and fate of cardiomyocytes after ischaemia/reperfusion

Lack of functional assembly in mitochondrial supercomplexes: a new insight into impaired mitochondrial function?

High-mobility group box 1 protein (HMGB1) in ischaemic heart disease: beneficial or deleterious?

New pathophysiological function of protein phosphatase 2A?

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Front matter/Back matter

Contents Page

Announcement: Spotlight Issue on Cardiac Protection

Announcement: Review Focus on Molecular Imaging in the Cardiovascular System

Noticias del día

Original Articles

Cardiac mitochondria in heart failure: decrease in respirasomes and oxidative phosphorylation

High-mobility group box 1 restores cardiac function after myocardial infarction in transgenic mice

Diastolic dysfunction in alveolar hypoxia: a role for interleukin-18-mediated increase in protein phosphatase 2A

Spatial non-uniformity of excitation–contraction coupling can enhance arrhythmogenic-delayed afterdepolarizations in rat cardiac muscle

The circadian pacemaker generates similar circadian rhythms in the fractal structure of heart rate in humans and rats

The PGE2-Stat3 interaction in doxorubicin-induced myocardial apoptosis

Decrease of peroxisome proliferator-activated receptor delta expression in cardiomyopathy of streptozotocin-induced diabetic rats

Porous tissue grafts sandwiched with multilayered mesenchymal stromal cell sheets induce tissue regeneration for cardiac repair

Overlapping and distinct roles for PI3K? and isoforms in S1P-induced migration of human and mouse endothelial cells

B2-kinin receptor plays a key role in B1-, angiotensin converting enzyme inhibitor-, and vascular endothelial growth factor-stimulated in vitro angiogenesis in the hypoxic mouse heart

Rho-GDP dissociation inhibitor alpha downregulated by low shear stress promotes vascular smooth muscle cell migration and apoptosis: a proteomic analysis

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Review

Remodelling of gap junctions and connexin expression in diseased myocardium

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